HemButikTabletter och kapslarMethyldrostanolone 10 mg

Methyldrostanolone 10 mg

10 mg · 100 tablets · Methyldrostanolone provides a highly potent anabolic reference standard for research on AR-driven protein synthesis, metabolic profiling, hepatotoxicity of 17-α-alkylated steroids, and comparative AAS analysis.

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Beskrivning

Methyldrostanolone 10 mg

Varumärke: Schweiziska botemedel
Category: Oral AAS Research Compound
Innehåll: 100 tablets | 10 mg per tablet (1,000 mg total)
Pris: €49
Tillverkad i: Switzerland


🧪 Produktöversikt

Methyldrostanolone 10 mg — commonly referenced as “Superdrol” in unregulated contexts — is a
synthetic anabolic–androgenic steroid (AAS) structurally derived from the dihydrotestosterone (DHT) family.
Its 17-α-alkylated modification grants oral bioavailability, making it a preferred laboratory reference compound
for studies involving androgen receptor (AR) activation, metabolic processing, toxicity profiling, and comparative
AAS analysis.

Methyldrostanolone is valued in research due to its strong anabolic properties, high potency, and well-defined
chemical identity. These attributes make it a useful benchmark in experiments requiring a consistent anabolic
signal or a known control compound for evaluating novel steroids and metabolites.


✅ Viktiga forskningsresultat

Strong anabolic transcriptional activity:
Acts as an AR agonist, stimulating protein synthesis, nitrogen retention, and anabolic gene expression.

Metabolic and toxicology profiling:
Its oral, methylated structure elevates hepatic workload, making it suitable for liver-stress and enzyme-induction models.

Comparative AAS evaluation:
Serves as a reference steroid in potency and toxicity benchmarking across DHT-derived and testosterone-derived analogs.

AR signaling studies:
Frequently used in cell-based and animal models for androgen receptor pathway research.


🧬 Föreslagna laboratorieapplikationer

  • Androgen receptor (AR) activation modeling
  • Protein synthesis and anabolic gene-expression studies
  • Comparative steroid potency and toxicology profiling
  • Liver-injury and hepatotoxicity research on oral 17-α-alkylated compounds
  • Metabolic analysis and steroid biotransformation pathways

📊 Tekniska specifikationer

Fält Value
Active Molecule Methyldrostanolone
Synonymer Methyl-DRO, Superdrol (unregulated contexts)
Chemical Class 17-α-alkylated DHT-derived anabolic–androgenic steroid (AAS)
Molecular Formula C21H34O2
Molekylvikt ~318.50 g/mol
CAS-nummer 3381-88-2
Primary Biological Target Androgen receptor (AR) agonist
Main Research Uses AR signaling, anabolic modeling, metabolic studies, toxicology, comparative AAS benchmarking
Formulation Oral tablets (10 mg)
Total Active Content 1,000 mg (100 × 10 mg)
Known Risk Domains Liver stress/toxicity, lipid imbalance (↓ HDL / ↑ LDL), cardiovascular strain, hormonal suppression
Regulatory Status Controlled/regulated AAS in many jurisdictions; WADA-prohibited category

❄️ PubChem-stödda forskningsinsikter

  • Binds the androgen receptor with strong anabolic transcriptional response
  • Promotes increased protein synthesis and nitrogen retention
  • 17-α-alkylation contributes to oral bioavailability and elevated hepatotoxic stress
  • Useful for comparing metabolic pathways of DHT-based steroids
  • Helpful in toxicology assays assessing oxidative and lipid metabolism alterations

⚠️ Dokumenterad forskning Biverkningar

  • Hepatic stress: enzyme elevation, cholestatic patterns, potential hepatotoxicity
  • Disrupted lipid metabolism (low HDL, elevated LDL)
  • Possible cardiovascular strain under prolonged exposures
  • Endocrine suppression of natural steroid biosynthesis
  • Hormonal dysregulation in chronic models

🧯 Hantering & efterlevnad

Endast för forskningsändamål — not for human or veterinary consumption.
Store in a dry, temperature-stable environment; protect from light.
Controlled or restricted status in many jurisdictions; ensure compliance before acquisition or use.
Suitable only for qualified laboratories conducting analytical, forensic, or experimental research.

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